High Blood Pressure Tips Treatments Calculating Target Heart Rate

Now, we’re ready to calculate your target heart rate. Here is the actual calculation. Target Heart Rate; THR. You’re going to take 220 minus your age, and you’re going to get a number there. You’re going to subtract your resting heart rate that we did in the earlier tutorial, and you’re going to get a number here. Then, based on how intensely you’re going to work out; if that’s a 60% intensity, or 70%, or even 80 or 85%, you?re going to multiply that by either .6, .7, .8, or .85. You’re going to get a number, and you’re going to add your resting heart rate to that number. This is an estimate of your actual target heart rate; it’s not the actual target heart rate. To do that you need more sophisticated.

Equipment. Here’s an example; a sixty year old man; 22060=160, and we’ll say that his resting heart rate is 60, as well, so you subtract that and we get 100. He’s going to workout at 60% of his max, so we get 60 there. Add the resting heart rate back on, so his target heart rate is 120. In the next tutorial we’re going to actually take an actor to his target heart rate, and we’re going to demonstrate how you do that. Once you get to your target heart rate, you want to keep it at that intensity for the duration of the exercise; thirty minutes; forty minutes; whatever your trainer says, and maintain it for that entire time.

Human Physiology Introduction to the Regulation of Mean Arterial Pressure

gt;gt; Ketchum: So to begin, we want to think about what can change your mean arterial pressure. MAP is the average pressure in the aorta during one cardiac cycle. Remember what MAP is equal to; it’s equal to cardiac output times total peripheral resistance, TPR. And cardiac output is equal to heart rate times stroke volume. So if I substitute, then, your equation for cardiac output (which is heart rate times stroke volume and that’s your cardiac output) and then I multiply by total peripheral resistance, I have yet another way to determine MAP. So how can we change MAP? Because if I change heart rate, if I change stroke volume or I change TPR, that will alter MAP. So how do we do that? We’re going to talk about two.

Different ways to do this. One of those is by increasing your cardiac output. So here this is an increase in cardiac output. If I increase CO, that means I have more blood leaving the heart. And when I have more blood leaving the heart –if this is the aorta, then we know when blood leaves the heart, when the ventricles contract, then that’s going to cause this aorta to expand outward. So here we’re increasing cardiac output, so that’s going increase the volume of blood in the aorta. Remember the walls are expanding in response to an increased cardiac output. And if we increase the volume of the blood in the aorta, that’s going to increase MAP. So just by increasing cardiac output. Now.

Please notice here that we did not change the total peripheral resistance. There’s no vasoconstriction or vasodilation in this instance; here we are only increasing cardiac output. Again, look at your formula. Cardiac output—if I increase this, that’s going to increase MAP. Now let’s try another option here, and that is let’s change your total peripheral resistance. Let’s increase it, but keep cardiac output constant. So here we have a constant cardiac output, but we are going to vasoconstrict these arterials. So if we vasoconstrict the arterials, this vasoconstriction, as we know, is going to increase total peripheral resistance.

So remember total peripheral resistance means the resistance across all of your arterials at one time. So if we increase total peripheral resistance, that’s going to increase MAP. So that gives us two ways to increase MAP. You increase cardiac output or you increased total peripheral resistance. How do we regulate MAP? So we’re going to be using neural and hormonal controls to do that. Here are the two ways to regulate MAP. So you have to keep MAP at a constant, right? You’ve got to maintain homeostasis; you don’t want it too high, you don’t want it too low. All right, so the two ways to do that are through shortterm regulation,.

Which occurs within seconds to minutes, and then there’s longterm regulation, and that occurs within minutes to days. All right, in shortterm regulation, this involves baroreceptors. It also involves chemoreceptors, but we won’t worry about those with respect to MAP. We talked about chemoreceptors with respect to respiration already. Baroreceptors are located in the carotid sinuses as well as the aortic arch. These baroreceptors are stretch receptors. So they are able to detect stretch, and then they will send action potentials to the cardiovascular control center in the medulla. Hopefully you’re asking yourself, “Well what will cause them to stretch?â€�.

Well, pressure. If we change the pressure in the vessels, then that can either increase stretch or decrease the amount of stretch. In longterm regulation it involves the kidneys. The kidneys, then, will regulate your blood volume, because if you regulate your blood volume, then we can regulate your mean arterial pressure.

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